HamsterHookah

Fantastic movie, but you are always in for a treat with Darren Aronofsky.

This is a broad generalization and certainly not the top reason we private school, but curious even if it were, what is inherently wrong with this in your view?

In what capacity and roles in the CPG food space, as specific as you can be or vague as you want, would be helpful to know what's informing your experience. I've spent a little bit in the space myself /greenlanternmeme

Are you seriously asking for a discussion and conversation when you ask this? Because in my experience, even with measured tone and reasoned responses, responding to questions like the below tends to just be bait to neg-bombed by the usual suspects for cathartic release.

Poor people eat trash for a variety of reasons and only some are by willful, explicit choice. The others appear to be choice, but are really informed by the 2/3 of the iceburg you don't see-- generational poverty, stress/illness induced by poverty that affects metabolism and health, systematic racism, aforementioned food desserts that drive convenience culture, the culture and view in and around food that is born around all these things, etc. I think you are being either ignorant or arrogant, by virtue of your virtue-- that is to say, because you once were fat, but now are not, your lived experience and view is very myopic. You were once fat and now are found, but without the amazing grace for fat people, it seems.

Yes, very sad and very funny. Oof.

Avocado toast and cigarettes are why people can't afford housing! /lol /sarcasm

Shows you the lightning-in-a-bottle, perfect-time-and-place, magic S1 had in that it was more than just a throwaway comedy show for most people, it struck an emotional nerve while being comedic. S2 was a severe disappointment and fall from that for some, including me, fairly or unfairly based on the expectations the show created in being able to be an emotionally compelling vehicle. I think it's too early to judge S3 and can't really judge ep1 except to say on it's own it was meh but may act to serve a greater role in making this season great through the set up. Let's watch and see!

Also, to add to your points, all the people who want to look at black and white photos of Spring Break 1980 and cut to Universal Studios 2023, imho, are failing to calculate the damage that has been done to the brain that controls things like weight and eating after years of abusing food. Food is a drug and it’s been abused and you have damaged people. It seems as simple as that. You can argue why society has become a bunch of food addicts and food injured people, and there are obvious answers like mass produced bad foods and fast food nation, but poverty and food desserts and cultural influence also play parts IMO.

The startling performance of software programs like ChatGPT has convinced some observers that machine consciousness is imminent; recently, a law in the U.K. recognized many animals, including crabs and lobsters, as sentient. From Humphrey’s point of view, these attitudes are misguided. Artificially intelligent machines are all perception, no sensation; they’ll never be sentient so long as they only process information. And animals such as reptiles and insects, which face little evolutionary pressure to develop a grasp of other minds, are also very unlikely to be sentient. If we don’t understand what sentience is for, we’re likely to see it everywhere. Conversely, once we perceive its practical value, we’ll acknowledge its rarity.

Listen-- I don't disagree with you at all. But, I have to know: How many margaritas have you had tonight?

Alrighty then. I'm not trying to sell you on it, I just found it interesting reading and an intersection between modern medicine and culture and this thread-- and think there is probably more to the story than just "fat people have no will power and discipline and/or lie about eating and are lazy" even as we find it to be less desirable and sub-optimal in ourselves and others to be overweight, bordering on a moral failure.

Did you miss this explanation of how/why it happens (allegedly)?: It's like saying, why are there more opiate addicts now than ever? Did their brains stop working from back in the 1900's? No, people are abusing food (fast food and sedentary nation) and injuring their brains in ways that they never used to (thus causing a negative physical dependency/outcome), is the prevailing theory.

Previous behaviors being ingesting 800 fewer calories? I mean, I guess they could be lying about that. Here is a different article talking about that: https://www.nytimes.com/2016/05/02/health/biggest-loser-weight-loss.html I mean, it's fine if you don't want to believe it because it's personal to you, but I'm just sharing some interesting research and writing about it, not attacking you. I congratulate you for your weight loss and sounds like you are doing great keeping it off (which is why you have a bit of indignation about all this).

And for @Wulaw Horn re: Bariatric (bolded below): Eli Lilly and Novo Nordisk together have at least twelve more obesity medications in development. Novo Nordisk reportedly spent about a hundred million dollars advertising Ozempic last year, and the two companies are spending roughly ten million dollars annually on lobbying. A primary focus of that lobbying is the proposed Treat and Reduce Obesity Act, which has been introduced in congressional sessions annually since 2012, and which would require Medicare to cover, among other treatments, chronic-weight-management drugs. Anticipating the passage of this bill within the next few years, Morgan Stanley has forecast that U.S. revenue from such drugs will increase four-hundredfold by the end of the decade. Obesity looks “set to become the next blockbuster pharma category,” it declared, in a report last year, which also predicted that social media and word of mouth will create an “exponential virtuous cycle” around the new medications: a quarter of people with obesity will seek treatment from physicians, up from the current seven per cent, and more than half of those who do will begin taking medicine. In March, WeightWatchers acquired the telehealth weight-loss company Sequence, which specializes in prescribing GLP-1 drugs. Controversially, the American Pediatric Association recently included weight-loss medication and bariatric surgery as part of a set of treatments that physicians should consider for kids with obesity. (Bariatric surgery, previously the only medical intervention that resulted in lasting weight loss for more than a small percentage of people, works in part because it, too, increases GLP-1 levels, and does so before any weight loss has occurred.) In clinical trials, patients who go off GLP-1 drugs regain much of their lost weight within a year. I asked Dr. Aronne, from Weill Cornell, about the possible medical consequences of irregular lifetime use, which seems to be a likely outcome for many patients, especially those who are prescribed the drugs at an early age. “That’s a great question,” he said, “and we don’t have the answer.” He suggested that doctors might begin treating obesity the way they treat hypertension. “You could start people on a tiny dose per week, and they would never get to the place where they have catastrophic problems,” he said. Patients would still need regular blood work and other monitoring; it’s likely enough that, as these drugs come into use in a wider patient population than ever before, new risks and complications will arise. But to Aronne, who has treated patients with serious health complications related to weight for thirty years, a lifetime on Wegovy seems far less dangerous than a lifetime of severe obesity.

From the article: "A healthy body can generally signal to the brain when it has had enough food. But that signalling system can be faulty, or get injured. “One of the most important things about obesity, and something most people don’t understand, is that, in the process of gaining weight, the neural circuitry of the brain that regulates weight is damaged,” Dr. Louis Aronne, the director of the Comprehensive Weight Control Center, at Weill Cornell Medicine, told me. (Aronne, like many other prominent practitioners of obesity medicine, has consulted on trials conducted by Novo Nordisk.) “The hypothalamus shows signs of inflammation and injury,” he went on. The prevailing theory, he explained, is that “too many calories coming in too quickly damages nerves that respond to the hormones that control body weight.” One of these hormones is leptin, which is produced in body fat, and which signals to the brain that it’s time to stop eating. But, if you gain fat, the oversupply of leptin can cause your body to be desensitized to it, making your brain erroneously believe that you are starving. “Your body tries to rebalance the system by slowing down the metabolism and increasing appetite,” Aronne said. After a person has gained enough weight to enter this cycle of metabolic misdirection, it becomes nearly impossible to lose that weight and keep it off long-term simply through diet and exercise. (About five per cent of people manage to do it.) A well-known study followed contestants on “The Biggest Loser,” the weight-loss-competition show, and found that the contestants’ metabolisms slowed so drastically after their weight loss that nearly all of them regained what they’d lost. One contestant, who’d dropped an astonishing two hundred and thirty-nine pounds, soon regained a hundred, and then began gaining weight whenever he ate more than eight hundred calories less than the average amount recommended for a man his size."

I know everyone has seen the word Ozempic as it has become a meme and it's going viral on social media the last few weeks/months culminating in The Oscar's, but there is a really great article about how the drug works and how being fat has a strong genetic component (to which the drug isolates/attacks): https://www.newyorker.com/magazine/2023/03/27/will-the-ozempic-era-change-how-we-think-about-being-fat-and-being-thin The drugs mimic a hormone called glucagon-like peptide-1, which stimulates insulin production and suppresses the production of glucagon, which raises blood sugar. The body naturally releases GLP-1 after a meal, and the hormone travels to the brain, triggering the feeling of fullness. GLP-1 drugs effectively inject that sense of satiety, and also slow the rate at which food empties out of the stomach; patients generally report a freedom from cravings and an inability to overeat without becoming ill. “I’m convinced that this basically replaced a signal my body has been missing my whole life,” a commenter in a Reddit group for people using semaglutide wrote recently. “All I can say,” a member of an online group called Lose the Fat wrote, “is that it is no wonder that skinny people think heavy people have no willpower. Their brains actually do tell them to stop eating. I had no idea.”

Might be the funniest thing I've heard in weeks. I died.

It's on netflix currently? I actually own the book and meant to read it but never have.

I re-read this book last year before watching. Greatness. I'm sad I am just now seeing this thread. Pure gold as always. Watched Thin Man a couple of months ago. One of my favorites.

Not according to Goldman Sachs, today, at least.

Thought this was interesting: Prices for an ad-free Disney+ subscription went up 38% last December, and yet the service — streaming home to Star Wars, Marvel, Pixar, and more — retained 94% of its subscribers, per The Wall Street Journal. Only 5% of users balked at the new $10.99/mo. charge and canceled. Less than 1% switched to the cheaper version with ads ($7.99/mo.). This doesn’t mean the ad-supported version isn’t working Data from Antenna shows the Disney+ “Basic” offering gaining traction. So far, 36% of new sign-ups have gone this less costly route. By comparison, adoption rates for HBO Max’s ad-bedazzled offering hover around 21%. Across its full streaming portfolio, Disney customers remain ever-increasing — Mickey and friends entered 2023 with 235.7m subs, per IndieWire. A Peter Pan-like dedication to youth helps: About half of Disney+ subscribers are families with children. All encouraging news for the people who really make dreams come true: Disney investors Streaming is an expensive game — the company segment housing Disney+ has lost nearly $10B since 2019, per WSJ. Widening its audience base and ad revenues through this new model is meant to help Disney close this gap. Plus: Existing Disney+ audiences not flinching at price increases may enable further bumps. (Case in point: $56 Disneyland tickets in 2005 vs. today’s $104 starting price. The park is still packed day in, day out.)

The better question, versus just the nominal amount of debt held at graduation, would be to factor in income and ability to service debt. So maybe a look at the debt held at n+interest after 10 years or something like that? E.g. a Pre-med/med school student loan obligation of $300k is different than an Education student loan of $120k. That said, the data I did find shows that the median debt load is manageable and serviceable (from 2023): https://educationdata.org/student-loan-debt-by-major E.g. RTF types are graduating with $26k (privilege?) where jobs as a gaffer are probably $40k.

Thanks for the insightful post. I too see the irony there and have a hard time trying to understand why the state would even want the administrative headache of HISD. There must be some good political football potential.

Yikes. There was a guy saying UTD where he sent his son was $40k/year.